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Mouse Models of Cerebral Arteriovenous Malformation.
Stroke; a journal of cerebral circulation, Jan-2016;47(1):293-300. Epub 2015 Sep 8.
Constitutively active Notch4 receptor elicits brain arteriovenous malformations through enlargement of capillary-like vessels.
Proceedings of the National Academy of Sciences of the United States of America, Dec-16-2014;111(50):18007-12. Epub 2014 Dec 2.
Endothelial ephrin-B2 is essential for arterial vasodilation in mice.
Microcirculation (New York, N.Y. : 1994), Oct-2014;21(7):578-86.
Deletion of Rbpj from postnatal endothelium leads to abnormal arteriovenous shunting in mice.
Development (Cambridge, England), Oct-2014;141(19):3782-92. Epub 2014 Sep 10.
Molecular identification of venous progenitors in the dorsal aorta reveals an aortic origin for the cardinal vein in mammals.
Development (Cambridge, England), Mar-2014;141(5):1120-8.
Notch4 is required for tumor onset and perfusion.
Vascular cell, 2013;[epublish]5(1):7.
Notch4 normalization reduces blood vessel size in arteriovenous malformations.
Science translational medicine, Jan-18-2012;4(117):117ra8.
Inefficient skeletal muscle repair in inhibitor of differentiation knockout mice suggests a crucial role for BMP signaling during adult muscle regeneration.
American journal of physiology. Cell physiology, May-2010;298(5):C1087-99. Epub 2010 Feb 24.
Constitutively active endothelial Notch4 causes lung arteriovenous shunts in mice.
American journal of physiology. Lung cellular and molecular physiology, Feb-2010;298(2):L169-77. Epub 2009 Nov 20.
Arterial-venous segregation by selective cell sprouting: an alternative mode of blood vessel formation.
Science (New York, N.Y.), Oct-9-2009;326(5950):294-8.
Endothelial Notch signaling is upregulated in human brain arteriovenous malformations and a mouse model of the disease.
Laboratory investigation; a journal of technical methods and pathology, Sep-2009;89(9):971-82. Epub 2009 Jun 22.
Cellular and molecular mechanism regulating blood flow recovery in acute versus gradual femoral artery occlusion are distinct in the mouse.
Journal of vascular surgery, Dec-2008;48(6):1546-58.
Artery and vein size is balanced by Notch and ephrin B2/EphB4 during angiogenesis.
Development (Cambridge, England), Nov-2008;135(22):3755-64.
Endothelial Notch4 signaling induces hallmarks of brain arteriovenous malformations in mice.
Proceedings of the National Academy of Sciences of the United States of America, Aug-5-2008;105(31):10901-6. Epub 2008 Jul 30.
c-myc in the hematopoietic lineage is crucial for its angiogenic function in the mouse embryo.
Development (Cambridge, England), Aug-2008;135(14):2467-77. Epub 2008 Jun 11.